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Specifics of anaphase chromatid segregation in Drosophila melanogaster mitotic mutants. / Lebedeva, L. I.; Fedorova, S. A.; Omelyanchuk, L. V.

In: Russian Journal of Genetics, Vol. 44, No. 8, 08.2008, p. 919-927.

Research output: Contribution to journal › Article › peer-review

Harvard

Lebedeva, LI, Fedorova, SA & Omelyanchuk, LV 2008, 'Specifics of anaphase chromatid segregation in Drosophila melanogaster mitotic mutants', Russian Journal of Genetics, vol. 44, no. 8, pp. 919-927. https://doi.org/10.1134/S102279540808005X

APA

Lebedeva, L. I., Fedorova, S. A., & Omelyanchuk, L. V. (2008). Specifics of anaphase chromatid segregation in Drosophila melanogaster mitotic mutants. Russian Journal of Genetics, 44(8), 919-927. https://doi.org/10.1134/S102279540808005X

Vancouver

Lebedeva LI, Fedorova SA, Omelyanchuk LV. Specifics of anaphase chromatid segregation in Drosophila melanogaster mitotic mutants. Russian Journal of Genetics. 2008 Aug;44(8):919-927. doi: 10.1134/S102279540808005X

Author

Lebedeva, L. I. ; Fedorova, S. A. ; Omelyanchuk, L. V. / Specifics of anaphase chromatid segregation in Drosophila melanogaster mitotic mutants. In: Russian Journal of Genetics. 2008 ; Vol. 44, No. 8. pp. 919-927.

BibTeX

@article{0ecd7f22438a42e483bd2681e6b10cf8,
title = "Specifics of anaphase chromatid segregation in Drosophila melanogaster mitotic mutants",
abstract = "Anaphase chromatid segregation defects (CSDs) were quantitatively and qualitatively studied in neural ganglion cells of third-instar larvae of several control wild type Drosophila melanogaster strains and four strains with mutations of the aar v158 , ff3, mast v40 , and CycB 2g cell cycle genes. A linear specificity was observed for the CSD frequency, type, determination, and correction probability. The probability of anaphase CSD correction was close to unity in the control strains and lower in the mutant strains. The lower correction probability in the mutant strains was explained in the context of two findings, that the mutations induced the CSDs that were atypical of the wild type strains and were potentially uncorrectable in anaphase and that the mutations negatively affected the relative anaphase time in mitosis.",
author = "Lebedeva, {L. I.} and Fedorova, {S. A.} and Omelyanchuk, {L. V.}",
year = "2008",
month = aug,
doi = "10.1134/S102279540808005X",
language = "English",
volume = "44",
pages = "919--927",
journal = "Russian Journal of Genetics",
issn = "1022-7954",
publisher = "PLEIADES PUBLISHING INC",
number = "8",

}

RIS

TY - JOUR

T1 - Specifics of anaphase chromatid segregation in Drosophila melanogaster mitotic mutants

AU - Lebedeva, L. I.

AU - Fedorova, S. A.

AU - Omelyanchuk, L. V.

PY - 2008/8

Y1 - 2008/8

N2 - Anaphase chromatid segregation defects (CSDs) were quantitatively and qualitatively studied in neural ganglion cells of third-instar larvae of several control wild type Drosophila melanogaster strains and four strains with mutations of the aar v158 , ff3, mast v40 , and CycB 2g cell cycle genes. A linear specificity was observed for the CSD frequency, type, determination, and correction probability. The probability of anaphase CSD correction was close to unity in the control strains and lower in the mutant strains. The lower correction probability in the mutant strains was explained in the context of two findings, that the mutations induced the CSDs that were atypical of the wild type strains and were potentially uncorrectable in anaphase and that the mutations negatively affected the relative anaphase time in mitosis.

AB - Anaphase chromatid segregation defects (CSDs) were quantitatively and qualitatively studied in neural ganglion cells of third-instar larvae of several control wild type Drosophila melanogaster strains and four strains with mutations of the aar v158 , ff3, mast v40 , and CycB 2g cell cycle genes. A linear specificity was observed for the CSD frequency, type, determination, and correction probability. The probability of anaphase CSD correction was close to unity in the control strains and lower in the mutant strains. The lower correction probability in the mutant strains was explained in the context of two findings, that the mutations induced the CSDs that were atypical of the wild type strains and were potentially uncorrectable in anaphase and that the mutations negatively affected the relative anaphase time in mitosis.

UR - http://www.scopus.com/inward/record.url?scp=50449103254&partnerID=8YFLogxK

U2 - 10.1134/S102279540808005X

DO - 10.1134/S102279540808005X

M3 - Article

AN - SCOPUS:50449103254

VL - 44

SP - 919

EP - 927

JO - Russian Journal of Genetics

JF - Russian Journal of Genetics

SN - 1022-7954

IS - 8

ER -

ID: 25439984