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Ontogeny of the Corticolimbic System and the Risk of Anxiety Disorders in Adolescence. / Dygalo, N. N.

в: Neuroscience and Behavioral Physiology, Том 52, № 2, 02.2022, стр. 277-286.

Результаты исследований: Научные публикации в периодических изданиях › статья › Рецензирование

Harvard

Dygalo, NN 2022, 'Ontogeny of the Corticolimbic System and the Risk of Anxiety Disorders in Adolescence', Neuroscience and Behavioral Physiology, Том. 52, № 2, стр. 277-286. https://doi.org/10.1007/s11055-022-01235-1

APA

Dygalo, N. N. (2022). Ontogeny of the Corticolimbic System and the Risk of Anxiety Disorders in Adolescence. Neuroscience and Behavioral Physiology, 52(2), 277-286. https://doi.org/10.1007/s11055-022-01235-1

Vancouver

Dygalo NN. Ontogeny of the Corticolimbic System and the Risk of Anxiety Disorders in Adolescence. Neuroscience and Behavioral Physiology. 2022 февр.;52(2):277-286. doi: 10.1007/s11055-022-01235-1

Author

Dygalo, N. N. / Ontogeny of the Corticolimbic System and the Risk of Anxiety Disorders in Adolescence. в: Neuroscience and Behavioral Physiology. 2022 ; Том 52, № 2. стр. 277-286.

BibTeX

@article{7580e7ded82948eaaae3d6bc8aed454e,
title = "Ontogeny of the Corticolimbic System and the Risk of Anxiety Disorders in Adolescence",
abstract = "Interactions of the amygdala, frontal cortex, and hippocampus, key structures in the corticolimbic system, play an important role in forming behavioral responses to threatening stimuli. The ontogenetic features of these interactions, both at the level of the formation of afferent and efferent connections between structures and the neurotransmitter and neurotrophic processes taking place within them, may be the causes of increases in the risk of psychoemotional disorders in the adolescent period as compared with earlier and later periods of life. Critical analysis of published data on this question is important for clarifying the mechanisms of formation of adolescent psychopathology and may aid the search for means of correcting it.",
keywords = "amygdala, anxiety, BDNF, brain-derived neurotrophic factor, chemogenetics, hippocampus, neurotransmitters, ontogeny, optogenetics, prefrontal cortex",
author = "Dygalo, {N. N.}",
note = "Publisher Copyright: {\textcopyright} 2022, Springer Nature Switzerland AG.",
year = "2022",
month = feb,
doi = "10.1007/s11055-022-01235-1",
language = "English",
volume = "52",
pages = "277--286",
journal = "Neuroscience and Behavioral Physiology",
issn = "0097-0549",
publisher = "Springer New York",
number = "2",

}

RIS

TY - JOUR

T1 - Ontogeny of the Corticolimbic System and the Risk of Anxiety Disorders in Adolescence

AU - Dygalo, N. N.

N1 - Publisher Copyright: © 2022, Springer Nature Switzerland AG.

PY - 2022/2

Y1 - 2022/2

N2 - Interactions of the amygdala, frontal cortex, and hippocampus, key structures in the corticolimbic system, play an important role in forming behavioral responses to threatening stimuli. The ontogenetic features of these interactions, both at the level of the formation of afferent and efferent connections between structures and the neurotransmitter and neurotrophic processes taking place within them, may be the causes of increases in the risk of psychoemotional disorders in the adolescent period as compared with earlier and later periods of life. Critical analysis of published data on this question is important for clarifying the mechanisms of formation of adolescent psychopathology and may aid the search for means of correcting it.

AB - Interactions of the amygdala, frontal cortex, and hippocampus, key structures in the corticolimbic system, play an important role in forming behavioral responses to threatening stimuli. The ontogenetic features of these interactions, both at the level of the formation of afferent and efferent connections between structures and the neurotransmitter and neurotrophic processes taking place within them, may be the causes of increases in the risk of psychoemotional disorders in the adolescent period as compared with earlier and later periods of life. Critical analysis of published data on this question is important for clarifying the mechanisms of formation of adolescent psychopathology and may aid the search for means of correcting it.

KW - amygdala

KW - anxiety

KW - BDNF

KW - brain-derived neurotrophic factor

KW - chemogenetics

KW - hippocampus

KW - neurotransmitters

KW - ontogeny

KW - optogenetics

KW - prefrontal cortex

UR - http://www.scopus.com/inward/record.url?scp=85126445331&partnerID=8YFLogxK

U2 - 10.1007/s11055-022-01235-1

DO - 10.1007/s11055-022-01235-1

M3 - Article

AN - SCOPUS:85126445331

VL - 52

SP - 277

EP - 286

JO - Neuroscience and Behavioral Physiology

JF - Neuroscience and Behavioral Physiology

SN - 0097-0549

IS - 2

ER -

ID: 35727036