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Targeting cancer cells through antibiotics-induced mitochondrial dysfunction requires autophagy inhibition
Research output
:
Contribution to journal
›
Article
›
peer-review
Free Radical Chemistry Laboratory
Section of Molecular Biology and Biotechnology
Overview
Cite this
DOI
https://doi.org/10.1016/j.canlet.2016.09.023
Final published version
Milan Esner
Dmitry Graifer
Matilde E. Lleonart
Alex Lyakhovich
Original language
English
Pages (from-to)
60-69
Number of pages
10
Journal
Cancer Letters
Volume
384
DOIs
https://doi.org/10.1016/j.canlet.2016.09.023
Publication status
Published -
1 Jan 2017
OECD FOS+WOS
Research areas
Antibiotics, Autophagy, Cancer, Mitochondria, Mitochondrial dysfunction, Mitophagy, Mitochondrial Degradation/drug effects, Humans, Autophagy/drug effects, Electron Transport Chain Complex Proteins/metabolism, Anti-Bacterial Agents/pharmacology, Antineoplastic Combined Chemotherapy Protocols/pharmacology, Microtubule-Associated Proteins/genetics, Transfection, Time Factors, Mitochondria/drug effects, Female, Cell Proliferation/drug effects, Breast Neoplasms/drug therapy, Reactive Oxygen Species/metabolism, Adenosine Triphosphate/metabolism, Signal Transduction/drug effects, Membrane Potential, Mitochondrial/drug effects, Drug Synergism, Energy Metabolism/drug effects, Cell Line, Tumor, Adenine/analogs & derivatives, FANCONI-ANEMIA CELLS, MANNER, RESISTANCE
ID: 9068822