Suppression of autophagy in the brain of transgenic mice with overexpression of А53Т-mutant α-synuclein as an early event at synucleinopathy progression

Research output: Contribution to journal › Article › peer-review

Animal model, Autophagy, LC3-II, Mouse, Parkinson's disease, А53Т-mutant α-synuclein, IMPAIRS MACROAUTOPHAGY, MODELS, CHAPERONE-MEDIATED AUTOPHAGY, EXPRESSION, A53T-mutant alpha-synuclein, PARKINSONS-DISEASE, Parkinson Disease/genetics, Autophagy/genetics, alpha-Synuclein/genetics, Substantia Nigra/metabolism, Disease Models, Animal, Tyrosine 3-Monooxygenase/metabolism, Mice, Transgenic, Dopaminergic Neurons/metabolism, Disease Progression, Frontal Lobe/metabolism, Corpus Striatum/metabolism, Animals, Mice

ID: 12155590